那位医学界好友给翻译下这段文字啊，感激不尽

Thus, the
phenotype and function of monocytes in human　sepsis, e.g., the reduced expression of HLA-DR and
costimulatory　molecules
and the increased expression of CD64, the reduced　production of inflammatory cytokines, and the
preserved　ROS
and NO generation, resemble those observed　in LPS-tolerant monocytes. These changes might indicate
the　reprogramming
of functions to control inflammatory damage　and preserve the ability to phagocytose and killinfecting　microorganisms.

Thus, the phenotype and function of monocytes in human sepsis, e.g., the reduced expression of HLA-DR and costimulatory molecules and the increased expression of CD64, the reduced　production of inflammatory cytokines, and the preserved ROS and NO generation, resemble those observed in LPS-tolerant monocytes. These changes might indicate the reprogramming of functions to control inflammatory damage　and preserve the ability to phagocytose and killinfecting　microorganisms.

因此，单核细胞在人类败血症，例如，HLA-DR的表达降低，表型和功能共刺激分子增加表达CD64，炎性细胞因子的产量减少，和保存ROS和NO的生成，类似于LPS容错单核细胞中所观察到的。这些变化可能表明重编程功能，以控制炎症损伤和保护的能力吞噬和killinfecting的微生物。

应该是这样的，有一些单词你不会是拼错了吧？

嗯我不是念医的, 只翻内容. 医学用语你自己查一下好了. 所以, 对於人体的 sepsis, monocytes 的功能和 phenotype (例如是, HLA-DR 和 costimulatory molecules 的表现下降, CD64 的表现上升, inclammatory cytokines 的生成减少, 以及 ROS 和 NO 的衍生被保留) 跟 LPS-tolerant monocytes 十分相似. 这些改变有可能表明, (楼主我猜你这里抄漏了一行) 透过功能重组以控制 inflammatory damage, 以及保留其 phagocytose 和 kill 受感染微生物之功效.

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